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Results 1-10 of 16 (Search time: 0.002 seconds).
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PreviewIssue DateTitleAuthor(s)
1997Enhanced adherence of Shiga-like toxin-producing Escherichia coli isolates from cases of human disease to intestinal epithelial (Henle 407) cellsPaton, A.; Voss, E.; Manning, P.; Paton, J.
1995Comparative toxicity and virulence of Escherichia coli clones expressing variant and chimeric Shiga-like toxin type II operonsPaton, A.; Bourne, A.; Manning, P.; Paton, J.
2008Cleavage of BiP by Subtilase CytotoxinPaton, A.; Paton, J.; Backer, M.; Backer, J.; Adelaide Research & Innovation
1998Translocated intimin receptors (Tir) of Shiga-toxigenic Escherichia coli isolates belonging to serogroups O26, O111, and O157 react with sera from patients with hemolytic-uremic syndrome and exhibit marked sequence heterogeneity.Paton, A.; Manning, P.; Woodrow, M.; Paton, J.; Barbieri, J.T.
1998Molecular analysis of shiga toxigenic Escherichia coli O111:H proteins which react with sera from patients with hemolytic-uremic syndrome.Voss, E.; Paton, A.; Manning, P.; Paton, J.
2015IRE1α is an endogenous substrate of endoplasmic-reticulum-associated degradationSun, S.; Shi, G.; Sha, H.; Ji, Y.; Han, X.; Shu, X.; Ma, H.; Inoue, T.; Gao, B.; Kim, H.; Bu, P.; Guber, R.; Shen, X.; Lee, A.; Iwawaki, T.; Paton, A.; Paton, J.; Fang, D.; Tsai, B.; Yates, J.; et al.
2011Cytoprotective roles of ERK and Akt in endoplasmic reticulum stress triggered by subtilase cytotoxinTian, T.; Zhao, Y.; Nakajima, S.; Huang, T.; Yao, J.; Paton, A.; Paton, J.; Kitamura, M.
2012BiP-mediated closing of the Sec61 channel limits Ca²⁺ leakage from the ERSchauble, N.; Lang, S.; Jung, M.; Cappel, S.; Schorr, S.; Ulucan, O.; Linxweiler, J.; Dudek, J.; Blum, R.; Helms, V.; Paton, A.; Paton, J.; Cavalie, A.; Zimmerman, R.
2012Escherichia coli 83972 expressing a P fimbriae oligosaccharide receptor mimic impairs adhesion of uropathogenic E. coliWatts, R.; Tan, C.; Ulett, G.; Carey, A.; Totsika, M.; Idris, A.; Paton, A.; Morona, R.; Paton, J.; Schembri, M.
2012The Escherichia coli subtilase cytotoxin A subunit specifically cleaves cell-surface GRP78 protein and abolishes COOH-terminal-dependent signalingRay, R.; de Ridder, G.; Eu, J.; Paton, A.; Paton, J.; Pizzo, S.

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