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Please use this identifier to cite or link to this item: https://hdl.handle.net/2440/47504
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dc.contributor.authorHankey, Graeme J.en
dc.contributor.authorEikelboom, J. W.en
dc.contributor.authorLoh, K.en
dc.contributor.authorYi, Q.en
dc.contributor.authorPizzi, J.en
dc.contributor.authorTang, M.en
dc.contributor.authorHickling, S.en
dc.contributor.authorLe, M.en
dc.contributor.authorKlijn, C. J. M.en
dc.contributor.authorDusitanond, P.en
dc.contributor.authorvan Bockxmeer, Frank M.en
dc.contributor.authorGelavis, A.en
dc.contributor.authorBaker, R.en
dc.contributor.authorJamrozik, Konraden
dc.date.issued2004en
dc.identifier.citationArteriosclerosis, Thrombosis, and Vascular Biology, 2004; 24 (8):147en
dc.identifier.issn1079-5642en
dc.identifier.urihttp://hdl.handle.net/2440/47504-
dc.description.statementofresponsibilityG.J. Hankey, J.W. Eikelboom, K. Loh, Q. Yi, J. Pizzi, M. Tang, S. Hickling, M. Le, C.J. M. Klijn, P. Dusitanond, F. van Bockxmeer, A. Gelavis, R. Baker and K. Jamroziken
dc.publisherLippincott Williams & Wilkinsen
dc.rights© 2004 American Heart Association, Inc.en
dc.titleIs there really a power shortage in clinical trials testing the "homocysteine hypothesis?".en
dc.typeJournal articleen
dc.contributor.schoolSchool of Population Health and Clinical Practiceen
dc.identifier.doi10.1161/01.ATV.0000136385.50973.68en
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